Rumpf HM, Gnauk J, Kirch HC, Esche H, Pützer BM.
Transformation-defective E1A-products of the Ad12-non-oncogenic host range mutant CS-1 are able to disrupt E2F-containing complexes and to transactivate E2F-motif containing promoters.
Biochem. Soc Transactions. 1996; 24:S510.
1996
Vorheriger Beitrag
Cell-density dependent growth reduction in interferon alpha-resistant U937 VR unravels different mechanisms of E2F(-1) inactivation.
Nächster Beitrag
Combination therapy with IL-2 and wild-type p53 expressed by Ad-vectors induces tumor regression in vivo without cytokine related toxicity.
Kontakt
Institut für Experimentelle Gentherapie und Tumorforschung
Core-Facility Virale Vektor & Genom-Editing Technologien
Biomedizinisches Forschungszentrum
Schillingallee 69
D-18057 Rostock
Sekretariat
Ingrid Winkler
(+49) 381 494-5066(+49) 381 494-5062
ingrid.winkler@med.uni-rostock.de
Department Leben, Licht & Materie
Forschungsbau LL&M
Albert-Einstein-Str. 25
D-18059 Rostock
Forschungsbau LL&M
Albert-Einstein-Str. 25
D-18059 Rostock